Tudóstér: Gönczi Mónika publikációi

Dienes, B., Szentesi, P., Szabó, L., Magyar, Z., Kovács, Z., Bazsó, T., Gönczi, M., Csernoch, L.: The contribution of PIEZO1 channels modifies our picture of skeletal muscle contraction.
Biophys. J. 123 (3_Suppl_1), 243a, 2024.

Gönczi, M., Teixeira, J., Barrera-Vilarmau, S., Mediani, L., Antoniani, F., Nagy, T., Fehér, K., Ráduly, Z., Ambrus, V., Tőzsér, J., Barta, E., Kövér, K., Csernoch, L., Carra, S., Fuxreiter, M.: Alternatively spliced exon regulates context-dependent MEF2D higher-order assembly during myogenesis.
Nat Comms. 14 1329, 2023.

Ráduly, Z., Szabó, L., Dienes, B., Szentesi, P., Bana, Á., Hajdú, T., Kókai, E., Hegedűs, C., Csernoch, L., Gönczi, M.: Migration of Myogenic Cells Is Highly Influenced by Cytoskeletal Septin7.
Cells. 12 (14), 1825, 2023.

Szabó, L., Telek, A., Fodor, J., Dobrosi, N., Dócs, K., Hegyi, Z., Gönczi, M., Csernoch, L., Dienes, B.: Reduced Expression of Septin7 Hinders Skeletal Muscle Regeneration.
Int. J. Mol. Sci. 24 (17), 13536, 2023.

Csernoch, L., Gönczi, M., Ráduly, Z., Szabó, L., Molnár, B., Szentesi, P., Dienes, B.: Simulated microgravity leads to altered intracellular calcium homeostasis and protein expression in C2C12 cultures.
Biophys. J. 122 (3_Suppl_1), 234a, 2023.

Gönczi, M., Csemer, A., Szabó, L., Sztretye, M., Fodor, J., Pocsai, K., Szenthe, K., Keller-Pintér, A., Köhler, Z., Nánási, P., Szentandrássy, N., Pál, B., Csernoch, L.: Astaxanthin Exerts Anabolic Effects via Pleiotropic Modulation of the Excitable Tissue.
Int. J. Mol. Sci. 23 (2), 917, 2022.

Singlár, Z., Ganbat, N., Szentesi, P., Osgonsandag, N., Szabó, L., Telek, A., Fodor, J., Dienes, B., Gönczi, M., Csernoch, L., Sztretye, M.: Genetic Manipulation of CB1 Cannabinoid Receptors Reveals a Role in Maintaining Proper Skeletal Muscle Morphology and Function in Mice.
Int. J. Mol. Sci. 23 (24), 1-21, 2022.

Gönczi, M., Ráduly, Z., Szabó, L., Fodor, J., Telek, A., Dobrosi, N., Balogh, N., Szentesi, P., Kis, G., Antal, M., Trencsényi, G., Dienes, B., Csernoch, L.: Septin7 is indispensable for proper skeletal muscle architecture and function.
eLife. 11 e75863, 2022.

Telek, A., Fodor, J., Dobrosi, N., Szabó, L., Gönczi, M., Dienes, B., Csernoch, L.: Septin-7 is indispensable in skeletal muscle regeneration.
J. Gen. Physiol. 154 (9), e2021ecc33, 2022.

Szabó, L., Balogh, N., Tóth, A., Angyal, Á., Gönczi, M., Csiki, D., Tóth, C., Balatoni, I., Jeney, V., Csernoch, L., Dienes, B.: The mechanosensitive Piezo1 channels contribute to the arterial medial calcification.
Front. Physiol. 13 1037230, 2022.

Dienes, C., Hézső, T., Kiss, D., Baranyai, D., Kovács, Z., Szabó, L., Magyar, J., Bányász, T., Nánási, P., Horváth, B., Gönczi, M., Szentandrássy, N.: Electrophysiological Effects of the Transient Receptor Potential Melastatin 4 Channel Inhibitor (4-Chloro-2-(2-chlorophenoxy)acetamido) Benzoic Acid (CBA) in Canine Left Ventricular Cardiomyocytes.
Int. J. Mol. Sci. 22 (17), 9499, 2021.

Gönczi, M., Dienes, B., Dobrosi, N., Fodor, J., Balogh, N., Oláh, T., Csernoch, L.: Septins, a cytoskeletal protein family, with emerging role in striated muscle.
J. Muscle Res. Cell Motil. 42 (2), 251-265, 2021.

Bachmann, M., Rossa, A., Antoniazzi, G., Biasutto, L., Carrer, A., Campagnaro, M., Leanza, L., Gönczi, M., Csernoch, L., Paradisi, C., Mattarei, A., Zoratti, M., Szabó, I.: Synthesis and cellular effects of a mitochondria-targeted inhibitor of the two-pore potassium channel TASK-3.
Pharmacol. Res. 164 105326, 2021.

Bachmann, M., Rossa, A., Antoniazzi, G., Biasutto, L., Carrer, A., Campagnaro, M., Leanza, L., Gönczi, M., Csernoch, L., Paradisi, C., Mattarei, A., Zoratti, M., Szabó, I.: Synthesis and cellular effects of a mitochondria-targeted inhibitor of the two-pore potassium channel TASK-3.
Eur. Biophys. J. Biophys. Lett. 50 (Suppl.), S105, 2021.

Gönczi, M., Fodor, J., Telek, A., Dobrosi, N., Ráduly, Z., Szabó, L., Szentesi, P., Dienes, B., Csernoch, L.: The Role of Septin7 in Skeletal Muscle Regeneration.
Biophys. J. 120 (3), 96a, 2021.

Sztretye, M., Singlár, Z., Szabó, L., Szentesi, P., Dienes, B., Gönczi, M., Csernoch, L.: Astaxanthin Improves Tetanic Force Without Altering Skeletal Muscle Excitation-Contraction Coupling in Mice.
Biophys. J. 118 (Suppl.), 408a, 2020.

Csernoch, L., Gönczi, M., Ráduly, Z., Szabó, L., Dobrosi, N., Szentesi, P., Dienes, B.: Essential Role of Septin 7 in Skeletal Muscle Structure and Function.
Biophys. J. 118 (Suppl.), 258a, 2020.

Gönczi, M., Szabó, L., Ráduly, Z., Dobrosi, N., Kis, G., Cseri, K., Dienes, B., Csernoch, L.: Mitochondrial Organization is Severly Modified in Skeletal Muscles of Septin 7 Knockdown Animals.
Biophys. J. 118 (Suppl.), 277a, 2020.

Sztretye, M., Dienes, B., Gönczi, M., Czirják, T., Csernoch, L., Dux, L., Szentesi, P., Keller-Pintér, A.: Astaxanthin: a Potential Mitochondrial-Targeted Antioxidant Treatment in Diseases and with Aging.
Oxidative Med. Cell. Longev. 2019 3849692, 2019.

Angyal, Á., Gönczi, M., Ráduly, Z., Szabó, L., Dobrosi, N., Dienes, B., Csernoch, L.: Septin 7 has an essential role in differentiation of C2C12 cells.
J. Muscle Res. Cell Motil. 40 (2), 242-243, 2019.

Dobrosi, N., Szabó, L., Ráduly, Z., Gönczi, M., Kis, G., Cseri, K., Dienes, B., Csernoch, L.: Septin 7 has no role in ec-coupling but severly modifies skeletal muscle architecture.
J. Muscle Res. Cell Motil. 40 (2), 250, 2019.

Nagy, L., Márton, J., Vida, A., Kis, G., Bokor, É., Kun, S., Gönczi, M., Docsa, T., Tóth, A., Antal, M., Gergely, P., Csóka, B., Pacher, P., Somsák, L., Bai, P.: Glycogen phosphorylase inhibition improves beta cell function.
Br. J. Pharmacol. 175 (2), 301-319, 2018.

Hegyi, B., Horváth, B., Váczi, K., Gönczi, M., Kistamás, K., Ruzsnavszky, F., Veress, R., Izu, L., Chen-Izu, Y., Bányász, T., Magyar, J., Csernoch, L., Nánási, P., Szentandrássy, N.: Ca2+-activated Cl- current is antiarrhythmic by reducing both spatial and temporal heterogeneity of cardiac repolarization.
J. Mol. Cell. Cardiol. 109 27-37, 2017.

Magyar, J., Horváth, B., Váczi, K., Hegyi, B., Gönczi, M., Dienes, B., Kistamás, K., Bányász, T., Baczkó, I., Varró, A., Seprényi, G., Csernoch, L., Nánási, P., Szentandrássy, N.: Calcium Activated Chloride Current in Mammalian Ventricular Myocytes.
Biophys. J. 112 (3), 36a, 2017.

Szentandrássy, N., Hegyi, B., Horváth, B., Váczi, K., Gönczi, M., Dienes, B., Kistamás, K., Veress, R., Ruzsnavszky, F., Bányász, T., Magyar, J., Baczkó, I., Varró, A., Seprényi, G., Csernoch, L., Nánási, P.: Recent advances in research of cardiac calcium-activated chloride channels.
Acta Physiol. 221 (S713), 22, 2017.

Gönczi, M., Nagy, D., Bai, P., Pál, B., Kis, G., Antal, M., Csernoch, L.: Role of TASK-3 channels in the mitochondria of melanoma cells.
Acta Physiol. 221 130, 2017.

Horváth, B., Váczi, K., Hegyi, B., Gönczi, M., Dienes, B., Kistamás, K., Bányász, T., Magyar, J., Baczkó, I., Varró, A., Seprényi, G., Csernoch, L., Nánási, P., Szentandrássy, N.: Sarcolemmal Ca2+-entry through L-type Ca2+ channels controls the profile of Ca2+-activated Cl- current in canine ventricular myocytes.
J. Mol. Cell. Cardiol. 97 125-139, 2016.

Gönczi, M., Nagy, Z., Nagy, D., Bai, P., Csernoch, L.: Investigation of metabolic processes in cultured melanoma cell lines.
Acta Physiol. 211 (Suppl.), 73, 2014.

Nagy, D., Gönczi, M., Dienes, B., Szöőr, Á., Fodor, J., Nagy, Z., Tóth, A., Fodor, T., Bai, P., Szűcs, G., Rusznák, Z., Csernoch, L.: Silencing the KCNK9 potassium channel (TASK-3) gene disturbs mitochondrial function, causes mitochondrial depolarization, and induces apoptosis of human melanoma cells.
Arch. Dermatol. Res. 306 (10), 885-902, 2014.

Nagy, D., Gönczi, M., Nagy, Z., Tóth, A., Dienes, B., Fodor, J., Szűcs, G., Rusznák, Z., Szöőr, Á., Csernoch, L.: Stable gene silencing of TASK-3 channels in melanoma cells induce intrinsic apoptosis.
Acta Physiol. 211 (Suppl.), 40, 2014.

Gönczi, M., Birinyi, P., Balázs, B., Szentandrássy, N., Harmati, G., Könczei, Z., Csernoch, L., Nánási, P.: Age-dependent changes in ion channel mRNA expression in canine cardiac tissues.
Gen. Physiol. Biophys. 31 (2), 153-162, 2012.

Kosztka, L., Rusznák, Z., Nagy, D., Nagy, Z., Fodor, J., Szűcs, G., Telek, A., Gönczi, M., Ruzsnavszky, O., Szentandrássy, N., Csernoch, L.: Inhibition of TASK-3 (KCNK9) channel biosynthesis changes cell morphology and decreases both DNA content and mitochondrial function of melanoma cells maintained in cell culture.
Melanoma Res. 21 (4), 308-322, 2011.

Ruzsnavszky, O., Telek, A., Gönczi, M., Balogh, A., Remenyik, É., Csernoch, L.: UV-B induced alteration in purinergic receptors and signaling on HaCaT keratinocytes.
J. Photochem. Photobiol. B. 105 (1), 113-118, 2011.

Füzi, M., Dienes, B., Szigeti, G., Németh, R., Vincze, J., Gönczi, M., Kertai, P., Csernoch, L.: Effect of fluvastatin on the calcium homeostasis of skeletal muscle fibers in hypercholesterolaemic rats.
Acta Physiol. Hung. 96 (1), 77-78, 2009.

Fodor, J., Matta, C., Juhász, T., Oláh, T., Gönczi, M., Szíjgyártó, Z., Gergely, P., Csernoch, L., Zákány, R.: Ionotropic purinergic receptor P2X4 is involved in the regulation of chondrogenesis in chicken micromass cell cultures.
Cell Calcium. 45 (5), 421-430, 2009.

Gönczi, M., Telek, A., Czifra, G., Balogh, A., Blumberg, P., Bíró, T., Csernoch, L.: Altered calcium handling following the recombinant overexpression of protein kinase C isoforms in HaCaT cells.
Exp. Dermatol. 17 (7), 584-591, 2008.

Fodor, J., Gönczi, M., Sztretye, M., Dienes, B., Oláh, T., Szabó, L., Csoma, E., Szentesi, P., Szigeti, G., Marty, I., Csernoch, L.: Altered expression of triadin 95 causes parallel changes in localized Ca2+ release events and global Ca2+ signals in skeletal muscle cells in culture.
J. Physiol. 586 (23), 5803-5818, 2008.

Rusznák, Z., Bakondi, G., Kosztka, L., Pocsai, K., Dienes, B., Fodor, J., Telek, A., Gönczi, M., Szűcs, G., Csernoch, L.: Mitochondrial expression of the two-pore domain TASK-3 channels in malignantly transformed and non-malignant human cells.
Virchows Arch. 452 (4), 415-426, 2008.

Gönczi, M., Szentandrássy, N., Fülöp, L., Telek, A., Szigeti, G., Magyar, J., Bíró, T., Nánási, P., Csernoch, L.: Hypotonic stress influence the membrane potential and alter the proliferation of keratinocytes in vitro.
Exp. Dermatol. 16 302-310, 2007.

Gönczi, M., Szentandrássy, N., Johnson, I., Heagerty, A., Weston, A.: Investigation of the role of TASK-2 channels in rat pulmonary arteries; pharmacological and functional studies following RNA interference procedures.
Br. J. Pharmacol. 147 (5), 496-505, 2006.

Pocsai, K., Kosztka, L., Bakondi, G., Gönczi, M., Fodor, J., Dienes, B., Szentesi, P., Kovács, I., Feniger-Barish, R., Kopf, E., Zharhary, D., Szűcs, G., Csernoch, L., Rusznák, Z.: Melanoma cells exhibit strong intracellular TASK-3-specific immunopositivity in both tissue sections and cell culture.
Cell. Mol. Life Sci. 63 (19-20), 2364-2376, 2006.

Szentesi, P., Szappanos, H., Szegedi, C., Gönczi, M., Jóna, I., Cseri, J., Kovács, L., Csernoch, L.: Altered elementary calcium release events and enhanced calcium release by thymol in rat skeletal muscle.
Biophys. J. 86 (3), 1436-1453, 2004.

Csernoch, L., Szappanos, H., Cseri, J., Gönczi, M., Sabatier, J., Altafaj, X., DeWaard, M., Ronjat, M.: Elementary calcium release events (ECRE) in the presence of the scorpion toxin maurocalcine.
Biophys. J. 86 579a, 2004.

Szappanos, H., Gönczi, M., Cseri, J., Kovács, L., Csernoch, L.: Elementary calcium release events (ECRE) in the presence of thymol on mammalian skeletal muscle.
J. Muscle Res. Cell Motil. 24 359, 2003.

Papp, H., Czifra, G., Lázár, J., Gönczi, M., Csernoch, L., Kovács, L., Bíró, T.: Protein kinase C isozymes regulate proliferation and high cell density-mediated differentiation in HaCaT keratinocytes.
Exp. Dermatol. 12 (6), 811-824, 2003.

Bakondi, E., Gönczi, M., Szabó, É., Bai, P., Pacher, P., Gergely, P., Kovács, L., Hunyadi, J., Szabó, C., Csernoch, L., Virág, L.: Role of Intracellular Calcium Mobilization and Cell-Density-Dependent Signaling in Oxidative-Stress-Induced Cytotoxicity in HaCaT Keratinocytes.
J. Invest. Dermatol. 121 (1), 88-95, 2003.

Gönczi, M., Papp, H., Kovács, L., Csernoch, L., Bíró, T.: Effect of protein kinase C on transmembrane calcium fluxes in HaCaT keratinocytes.
Exp. Dermatol. 11 (1), 25-33, 2002.

Gönczi, M., Papp, H., Bodó, E., Kovács, L., Bíró, T., Csernoch, L.: Effects of recombinant overexpression of PKCα and [delta] on receptor-coupled calcium handling in HaCaT keratinocytes.
J. Invest. Dermatol. 119 (3), 746, 2002.

Gönczi, M., Szigeti, G., Fülöp, L., Magyar, J., Kovács, L., Csernoch, L.: Role of stretch-activated channels in human keratinocytes.
Acta Physiol. Hung. 89 (Suppl.), 48, 2002.

Szentandrássy, N., Gönczi, M., Papp, H., Lázár, J., Kovács, J., Bíró, T., Csernoch, L.: Stretch-activated channels and their regulation by protein kinase C in HaCaT keratinocytes.
J. Invest. Dermatol. 119 (3), 746, 2002.