Tudóstér: Nagy Lilla Nikoletta publikációi

Fonódi, M., Nagy, L., Boratkó, A.: Role of protein phosphatases in tumor angiogenesis: assessing PP1, PP2A, PP2B and PTPs activity.
Int. J. Mol. Sci. 25 1-43, 2024.

Nagy, L., Rauch, B., Szerafin, T., Uray, K., Tóth, A., Bai, P.: Nicotinamide-riboside shifts the differentiation of human primary white adipocytes to beige adipocytes impacting substrate preference and uncoupling respiration through SIRT1 activation and mitochondria-derived reactive species production.
Front. Cell. Dev. Biol. 10 1-12, 2022.

Nagy, L., Béke, F., Juhász, L., Kovács, T., Juhász-Tóth, É., Docsa, T., Tóth, A., Gergely, P., Somsák, L., Bai, P.: Glycogen phosphorylase inhibitor, 2,3-bis[(2E)-3-(4-hydroxyphenyl)prop-2-enamido] butanedioic acid (BF142), improves baseline insulin secretion of MIN6 insulinoma cells.
PLoS One. 15 (9), 1-15, 2020.

Lódi, M., Priksz, D., Fülöp, G., Bódi, B., Gyöngyösi, A., Nagy, L., Kovács, Á., Kertész, A., Kocsis, J., Édes, I., Csanádi, Z., Czuriga, I., Kisvárday, Z., Juhász, B., Lekli, I., Bai, P., Tóth, A., Papp, Z., Czuriga, D.: Advantages of prophylactic versus conventionally scheduled heart failure therapy in an experimental model of doxorubicin-induced cardiomyopathy.
J. Transl. Med. 17 229, 2019.

Nagy, L., Rauch, B., Balla, N., Ujlaki, G., Kis, G., Omar, A., Kristóf, E., Sipos, A., Antal, M., Tóth, A., Debreceni, T., Horváth, A., Maros, T., Csizmadia, P., Szerafin, T., Bai, P.: Olaparib induces browning of in vitro cultures of human primary white adipocytes.
Biochem. Pharmacol. 167 76-85, 2019.

Nagy, L., Márton, J., Vida, A., Kis, G., Bokor, É., Kun, S., Gönczi, M., Docsa, T., Tóth, A., Antal, M., Gergely, P., Csóka, B., Pacher, P., Somsák, L., Bai, P.: Glycogen phosphorylase inhibition improves beta cell function.
Br. J. Pharmacol. 175 (2), 301-319, 2018.

Bai, P., Nagy, L., Abdul-Rahman, O., Rauch, B., Balla, N., Kristóf, E., Tóth, A., Szerafin, T.: NAD+ homeostasis plays role in mitochondrial biogenesis during beige adipocyte differentiation.
Biochim. Biophys. Acta. Bioenerg. 1859 e36, 2018.

Márton, J., Fodor, T., Nagy, L., Vida, A., Kis, G., Brunyánszki, A., Antal, M., Lüscher, B., Bai, P.: PARP10 (ARTD10) modulates mitochondrial function.
PLoS One. 13 (1), e0187789, 2018.

Abdul-Rahman, O., Kristóf, E., Doan-Xuan, Q., Vida, A., Nagy, L., Horváth, A., Simon, J., Maros, T., Szentkirályi, I., Palotás, L., Debreceni, T., Csizmadia, P., Szerafin, T., Fodor, T., Szántó, M., Tóth, A., Kiss, B., Bacsó, Z., Bai, P.: AMP-Activated Kinase (AMPK) Activation by AICAR in Human White Adipocytes Derived from Pericardial White Adipose Tissue Stem Cells Induces a Partial Beige-Like Phenotype.
PLoS One. 11 (6), e0157644, 2016.

Fodor, T., Szántó, M., Abdul-Rahman, O., Nagy, L., Dér, Á., Kiss, B., Bai, P.: Combined Treatment of MCF-7 Cells with AICAR and Methotrexate, Arrests Cell Cycle and Reverses Warburg Metabolism through AMP-Activated Protein Kinase (AMPK) and FOXO1.
PLoS One. 11 (2), 1-16, 2016.

Bai, P., Nagy, L., Fodor, T., Liaudet, L., Pacher, P.: Poly(ADP-ribose) polymerases as modulators of mitochondrial activity.
Trends Endocrinol. Metab. 26 (2), 75-83, 2015.

Szántó, M., Brunyánszki, A., Márton, J., Vámosi, G., Nagy, L., Fodor, T., Kiss, B., Virág, L., Gergely, P., Bai, P.: Deletion of PARP-2 induces hepatic cholesterol accumulation and decrease in HDL levels.
Biochim. Biophys. Acta. Mol. Basis Dis. 1842 (4), 594-602, 2014.

Nagy, L., Docsa, T., Szántó, M., Brunyánszki, A., Hegedűs, C., Márton, J., Kónya, B., Virág, L., Somsák, L., Gergely, P., Bai, P.: Glycogen Phosphorylase Inhibitor N-(3,5-Dimethyl-Benzoyl)-N'-(β-D-Glucopyranosyl)Urea Improves Glucose Tolerance under Normoglycemic and Diabetic Conditions and Rearranges Hepatic Metabolism.
PLoS One. 8 (7), e69420-, 2013.

Szántó, M., Brunyánszki, A., Kiss, B., Nagy, L., Gergely, P., Virág, L., Bai, P.: Poly(ADP-ribose) polymerase-2: emerging transcriptional roles of a DNA-repair protein.
Cell. Mol. Life Sci. 69 (24), 4079-4092, 2012.