Tudóstér: Bekéné Debreceni Ildikó publikációi

Kappelmayer, J., Bekéné Debreceni, I., Fejes, Z., Nagy, B.: Inflammation, Sepsis, and the Coagulation System.
Hamostaseologie. 44 (4), 268-276, 2024.

Ghansah, H., Orbán-Kálmándi, R., Bekéné Debreceni, I., Katona, É., Rejtő, L., Váróczy, L., Lóczi, L., Laat, B., Huskens, D., Kappelmayer, J., Bagoly, Z.: Low factor XIII levels and altered fibrinolysis in patients with multiple myeloma.
Thromb. Res. 234 12-20, 2024.

Gonda, L., Torner, B., Ghansah, H., Bekéné Debreceni, I., Váróczy, L., Pénzes-Daku, K., Kappelmayer, J.: Monoclonal whole IgG impairs both fibrin and thrombin formation: hemostasis and surface plasmon resonance studies.
Clin. Chem. Lab. Med. 62 (9), 1863-1869, 2024.

Krajcsir, B., Pócsi, M., Fejes, Z., Nagy, B., Kappelmayer, J., Bekéné Debreceni, I.: Ponatinib Induces a Procoagulant Phenotype in Human Coronary Endothelial Cells via Inducing Apoptosis.
Pharmaceutics. 16 (4), 1-14, 2024.

Ghansah, H., Bekéné Debreceni, I., Váróczy, L., Rejtő, L., Lóczi, L., Bagoly, Z., Kappelmayer, J.: Patients with multiple myeloma and monoclonal gammopathy of undetermined significance have variably increased thrombin generation and different sensitivity to the anticoagulant effect of activated protein C.
Thromb. Res. 223 44-52, 2023.

Illési, Á., Fejes, Z., Pócsi, M., Bekéné Debreceni, I., Hódosi, K., Nagy, B., Kappelmayer, J., Kőszegi, Z., Csanádi, Z., Szűk, T.: Technically Challenging Percutaneous Interventions of Chronic Total Occlusions Are Associated with Enhanced Platelet Activation.
J Clin Med. 12 (21), 1-14, 2023.

Somodi, L., Bekéné Debreceni, I., Kis, G., Cozzolino, M., Kappelmayer, J., Antal, M., Panyi, G., Bárdos, H., Mutch, N., Muszbek, L.: Activation mechanism dependent surface exposure of cellular factor XIII on activated platelets and platelet microparticles.
J. Thromb. Haemost. 20 (5), 1223-1235, 2022.

Illési, Á., Bekéné Debreceni, I., Fejes, Z., Nagy, B., Hódosi, K., Kappelmayer, J., Csanádi, Z., Szűk, T.: Effect of invasive therapeutic coronary interventions on endothelial cell activation and thrombin generation in patients with chronic total coronary occlusion.
Thromb. Res. 217 64-72, 2022.

Szabó, G., Bekéné Debreceni, I., Tarr, T., Soltész, P., Osterud, B., Kappelmayer, J.: Anti-[beta]2-glycoprotein I autoantibodies influence thrombin generation parameters via various mechanisms.
Thromb. Res. 197 124-131, 2021.

Mezei, G., Batár, P., Kozma, L., Illés, Á., Kappelmayer, J., Bekéné Debreceni, I.: Ponatinib Exerts an Inhibitory Effect on Collagen-induced Platelet Aggregation and Generation of Coated-Platelets.
Anticancer Res. 41 (10), 4867-4874, 2021.

Ghansah, H., Bekéné Debreceni, I., Fejes, Z., Nagy, B., Kappelmayer, J.: The Proteasome Inhibitor Bortezomib Induces Apoptosis and Activation in Gel-Filtered Human Platelets.
Int. J. Mol. Sci. 22 (16), 1-13, 2021.

Orosz, Z., Bárdos, H., Shemirani, A., Bekéné Debreceni, I., Lassila, R., Riikonen, A., Kremer Hovinga, J., Seiler, T., van Dorland, H., Schroeder, V., Boda, Z., Nemes, L., Früh Eppstein, B., Nagy, B., Facskó, A., Kappelmayer, J., Muszbek, L.: Cellular Factor XIII, a Transglutaminase in Human Corneal Keratocytes.
Int. J. Mol. Sci. 20 (23), 1-17, 2019.

Mezei, G., Bekéné Debreceni, I., Kerényi, A., Reményi, G., Szász, R., Illés, Á., Kappelmayer, J., Batár, P.: Dasatinib inhibits coated-platelet generation in patients with chronic myeloid leukemia.
Platelets. 30 (7), 836-843, 2019.

Bekéné Debreceni, I., Mezei, G., Batár, P., Illés, Á., Kappelmayer, J.: Dasatinib Inhibits Procoagulant and Clot Retracting Activities of Human Platelets.
Int. J. Mol. Sci. 20 (21), 1-14, 2019.

Gyöngyösi, A., Szőke, K., Fenyvesi, F., Fejes, Z., Bekéné Debreceni, I., Nagy, B., Tósaki, Á., Lekli, I.: Inhibited autophagy may contribute to heme toxicity in cardiomyoblast cells.
Biochem. Biophys. Res. Commun. 511 (4), 732-738, 2019.

Bekéné Debreceni, I., Szász, R., Kónya, Z., Erdődi, F., Kiss, F., Kappelmayer, J.: L-Selectin Expression Is Influenced by Phosphatase Activity in Chronic Lymphocytic Leukemia.
Cytom. Part B-Clin. Cytom. 96 (2), 149-157, 2019.

Tóth, J., Bekéné Debreceni, I., Deák, Á., Pető, K., Berhés, M., Hajdu, E., Szabó, J., Németh, N., Fülesdi, B., Kappelmayer, J.: Characteristics of thrombin generation in a fulminant in a fulminant porcine sepsis model.
Thromb. Res. 158 25-34, 2017.

Kerényi, A., Bekéné Debreceni, I., Oláh, Z., Ilonczai, P., Bereczky, Z., Nagy, B., Muszbek, L., Kappelmayer, J.: Evaluation of flow cytometric HIT assays in relation to an IgG-specific immunoassay and clinical outcome.
Cytom. Part B-Clin. Cytom. 92 (5), 389-397, 2017.

Fejes, Z., Póliska, S., Czimmerer, Z., Káplár, M., Penyige, A., Gál Szabó, G., Bekéné Debreceni, I., Kunapuli, S., Kappelmayer, J., Nagy, B.: Hyperglycemia suppresses microRNA expression in platelets to increase P2RY12 and SELP levels in type 2 diabetes mellitus.
Thromb. Haemost. 117 (3), 529-542, 2017.

Hudák, R., Bekéné Debreceni, I., Deák, I., Gál Szabó, G., Hevessy, Z., Antal-Szalmás, P., Osterud, B., Kappelmayer, J.: Laboratory characterization of leukemic cell procoagulants.
Clin. Chem. Lab. Med. 55 (8), 1215-1223, 2017.

Tóth, J., Bekéné Debreceni, I., Berhés, M., Hajdu, E., Deák, Á., Pető, K., Szabó, J., Németh, N., Fülesdi, B., Kappelmayer, J.: Red blood cell and platelet parameters are sepsis predictors in an Escherichia coli induced lethal porcine modell.
Clin. Hemorheol. Microcirc. 66 (3), 249-259, 2017.

Hudák, R., Vincze, J., Csernoch, L., Bekéné Debreceni, I., Oláh, T., Erdődi, F., Clemetson, K., Kappelmayer, J.: The phosphatase inhibitor calyculin-A impairs clot retraction, platelet activation and thrombin generation.
Biomed Res. Int. 2017 1-10, 2017.

Becs, G., Hudák, R., Fejes, Z., Bekéné Debreceni, I., Bhattoa, H., Balla, J., Kappelmayer, J.: Haemodiafiltration elicits less platelet activation compared to haemodialysis.
BMC Nephrol. 17 (1), 147, 2016.

Szűk, T., Fejes, Z., Bekéné Debreceni, I., Kerényi, A., Édes, I., Kappelmayer, J., Nagy, B.: Integrity bare-metal coronary stent-induced platelet and endothelial cell activation results in a higher risk of restenosis compared to Xience everolimus-eluting stents in stable angina patients.
Platelets. 27 (5), 410-419, 2016.

Reményi, G., Szász, R., Bekéné Debreceni, I., Szarvas, M., Batár, P., Nagy, B., Kappelmayer, J., Udvardy, M.: Comparison of coated-platelet levels in patients with essential thrombocythemia with and without hydroxyurea treatment.
Platelets. 24 (6), 486-492, 2013.

Kappelmayer, J., Bekéné Debreceni, I., Vida, A., Antal-Szalmás, P., Clemetson, K., Nagy, B.: Distinct effects of Re- and S-forms of LPS on modulating platelet activation.
J. Thromb. Haemost. 11 (4), 775-778, 2013.

Nagy, B., Bekéné Debreceni, I., Kappelmayer, J.: Flow cytometric investigation of classical and alternative platelet activation markers.
EJIFCC. 23 (4), 124-134, 2013.

Antal-Szalmás, P., Nagy, B., Bekéné Debreceni, I., Kappelmayer, J.: Measurement of soluble biomarkers by flow cytometry.
EJIFCC. 23 (4), [1-8], 2013.

Miszti-Blasius, K., Bekéné Debreceni, I., Felszeghy, S., Dezső, B., Kappelmayer, J.: Lack of P-selectin glycoprotein ligand-1 protects mice from thrombosis after collagen/epinephrine challenge.
Thromb. Res. 127 (3), 228-234, 2011.

Nagy, B., Szűk, T., Bekéné Debreceni, I., Kappelmayer, J.: Platelet-derived microparticle levels are significantly elevated in patients treated by elective stenting compared to subjects with diagnostic catheterization alone.
Platelets. 21 (2), 147-151, 2010.

Simon, Z., Kiss, A., Erdődi, F., Setiadi, H., Bekéné Debreceni, I., Nagy, B., Kappelmayer, J.: Protein phosphatase inhibitor calyculin-A modulates activation markers in TRAP-stimulated human platelets.
Platelets. 21 (7), 555-562, 2010.

Ivády, G., Bekéné Debreceni, I., Kissné, S., Hevessy, Z., Kappelmayer, J.: A timidin kináz aktivitás összehasonlító elemzése egyéb prognosztikai markerekkel krónikus lymphocytás leukémiában.
Klin. Kísérl. Lab. Med. 33 (2), 7-11, 2008.