Tudóstér: OTKA-116228 publikációi

Balogh, G., Bereczky, Z.: The Interaction of Factor Xa and IXa with Non-Activated Antithrombin in Michaelis Complex: Insights from Enhanced-Sampling Molecular Dynamics Simulations.
Biomolecules. 13 (5), 1-21, 2023.

Gindele, R., Kerényi, A., Kállai, J., Pfliegler, G., Schlammadinger, Á., Szegedi, I., Major, T., Szabó, Z., Bagoly, Z., Kiss, C., Kappelmayer, J., Bereczky, Z.: Resolving Differential Diagnostic Problems in von Willebrand Disease, in Fibrinogen Disorders, in Prekallikrein Deficiency and in Hereditary Hemorrhagic Telangiectasia by Next-Generation Sequencing.
Life (Basel). 11 (3), 1-23, 2021.

Balogh, G., Komáromi, I., Bereczky, Z.: The mechanism of high affinity pentasaccharide binding to antithrombin, insights from Gaussian accelerated molecular dynamics simulations.
J. Biomol. Struct. Dyn. 38 (16), 4718-4732, 2020.

Kovac, M., Mitic, G., Mikovic, Z., Mandic, V., Miljic, P., Mitrovic, M., Tomic, B., Bereczky, Z.: The influence of specific mutations in the AT gene (SERPINC1) on the type of pregnancy related complications.
Thromb. Res. 173 12-19, 2019.

Kovac, M., Mitic, G., Lalic-Cosic, S., Djordjevic, V., Tomic, B., Muszbek, L., Bereczky, Z.: Evaluation of endogenous thrombin potential among patients with antithrombin deficiency.
Thromb. Res. 166 50-53, 2018.

Bereczky, Z., Oláh, Z., Ajzner, É., Kappelmayer, J.: Az új orális antikoagulánsokkal történő kezelés laboratóriumi vonatkozásai.
Orvosi Hetilap. 158 (49), 1930-1945, 2017.

Gindele, R., Selmeczi, A., Oláh, Z., Ilonczai, P., Pfliegler, G., Marján, E., Nemes, L., Nagy, Á., Losonczy, H., Mitic, G., Kovac, M., Balogh, G., Komáromi, I., Schlammadinger, Á., Molnárné Rázsó, K., Boda, Z., Muszbek, L., Bereczky, Z.: Clinical and laboratory characteristics of antithrombin deficiencies: a large cohort study from a single diagnostic center.
Thromb. Res. 160 119-128, 2017.

Major, T., Gindele, R., Szabó, Z., Alef, T., Thiele, B., Bora, L., Kis, Z., Bárdossy, P., Rácz, T., Havacs, I., Bereczky, Z.: Evidence for the founder effect of a novel ACVRL1 splice-site mutation in Hungarian hereditary hemorrhagic telangiectasia families.
Clin. Genet. 90 (5), 466-467, 2016.