Nagy László

Bódi, B., Pásztorné Tóth, E., Nagy, L., Tóth, A., Mártha, L., Kovács, Á., Balla, G., Kovács, T., Papp, Z.: Titin isoforms are increasingly protected against oxidative modifications in developing rat cardiomyocytes.
Free Radic. Biol. Med. 113, 224-235, 2017.

doi DEA

Folyóirat-mutatók:

Q1 Biochemistry

Q1 Physiology (medical)

2016

Nagy, L., Pollesello, P., Haikala, H., Végh, Á., Sorsa, T., Levijoki, J., Szilágyi, S., Édes, I., Tóth, A., Papp, Z., Papp, G. J.: ORM-3819 promotes cardiac contractility through Ca2+ sensitization in combination with selective PDE III inhibition, a novel approach to inotropy.
Eur. J. Pharmacol. 775, 120-129, 2016.

doi DEA

Folyóirat-mutatók:

Q2 Pharmacology

2015

Nagy, L., Kovács, Á., Bódi, B., Pásztorné Tóth, E., Fülöp, G. Á., Tóth, A., Édes, I., Papp, Z.: The novel cardiac myosin activator omecamtiv mecarbil increases the calcium sensitivity of force production in isolated cardiomyocytes and skeletal muscle fibres of the rat.
Br. J. Pharmacol. 172 (18), 4506-4518, 2015.

doi DEA

Folyóirat-mutatók:

D1 Pharmacology

2014

Kovács, Á., Papp, Z., Nagy, L.: Causes and pathophysiology of heart failure with preserved ejection fraction.
Heart Fail. Clin. 10 (3), 389-398, 2014.

doi DEA

Folyóirat-mutatók:

Q2 Cardiology and Cardiovascular Medicine

Q2 Medicine (miscellaneous)

Nagy, L., Pollesello, P., Papp, Z.: Inotropes and inodilators for acute heart failure: sarcomere active drugs in focus.
J. Cardiovasc. Pharmacol. 64 (3), 199-208, 2014.

doi DEA

Folyóirat-mutatók:

Q1 Cardiology and Cardiovascular Medicine

Q2 Pharmacology

Balogh, Á., Tóth, A., Pásztorné Tóth, E., Nagy, L., Kovács, Á., Kalász, J., Contreras, G. A., Édes, I., Papp, Z.: Myofilament carbonylation modulates contractility in human cardiomyocytes.
Exp. Clin. Cardiol. 20 (1), 2026-2035, 2014.

DEA

Folyóirat-mutatók:

Q3 Cardiology and Cardiovascular Medicine

Q4 Physiology

Q3 Physiology (medical)