Szántó, M., Yélamos, J., Bai, P.: Specific and shared biological functions of PARP2-is PARP2 really a lil' brother of PARP1?.
Expert Rev. Mol. Med. 26 1-21, 2024.

Antal, D., Pór, Á., Kovács, I., Dull, K., Póliska, S., Ujlaki, G., Demény, M., Szöllősi, A., Kiss, B., Szegedi, A., Bai, P., Szántó, M.: PARP2 promotes inflammation in psoriasis by modulating estradiol biosynthesis in keratinocytes.
J. Mol. Med. (Berl). 101 (8), 987-999, 2023.

Antal, D., Pór, Á., Kovács, I., Dull, K., Póliska, S., Ujlaki, G., Demény, M., Szöllősi, A., Kiss, B., Szegedi, A., Bai, P., Szántó, M.: PARP2 promotes inflammation in sporiasis by modulating estradiol biosynthesis in keratinocytes.
In: FEBS Advanced Lecture Course: PARP 2023: Research on the family of poly(ADP-ribose) polymerases; Book of Abstracts. Ed.: Andreja Mikoc, Ivan Matic, Ruder Boskovic Institue, Hvar, 60, 2023. ISBN: 9789537941468

Antal, D., Alimohammadi, S., Bai, P., Szöllősi, A., Szántó, M.: Antigen-Presenting Cells in Psoriasis.
Life (Basel). 12 (2), 1-10, 2022.

Antal, D., Janka, E., Szabó, J., Szabó, I., Szegedi, A., Gáspár, K., Bai, P., Szántó, M.: Culture-based analyses of skin bacteria in lesional moist, and unaffected dry and sebaceous skin regions of hidradenitis suppurativa patients.
J. Eur. Acad. Dermatol. Venereol. 36 (9), e731-e733, 2022.

Antal, D., Pór, Á., Dull, K., Kovács, I., Szegedi, A., Bai, P., Szántó, M.: Poly(ADP-ribose) polymerase (PARP)-2 promotes inflammation in psoriasis by regulating estradiol biosynthesis in keratinocytes.
J. Invest. Dermatol. 142 (12), S237, 2022.

Szántó, M., Gupte, R., Kraus, W., Pacher, P., Bai, P.: PARPs in lipid metabolism and related diseases.
Prog. Lipid Res. 84 1-24, 2021.

Kiss, B., Szántó, M., Hegedűs, C., Antal, D., Szödényi, A., Márton, J., Méhes, G., Virág, L., Szegedi, A., Bai, P.: Poly(ADP-ribose) polymerase-1 depletion enhances the severity of inflammation in an imiquimod-induced model of psoriasis.
Exp. Dermatol. 29 (1), 79-85, 2020.

Jankó, L., Sári, Z., Kovács, T., Kis, G., Szántó, M., Antal, M., Juhász, A., Bai, P.: Silencing of PARP2 blocks autophagic degradation.
Cells. 9 (2), 1-21, 2020.

Szántó, M., Bai, P.: The role of ADP-ribose metabolism in metabolic regulation, adipose tissue differentiation and metabolism.
Genes Dev. 34 (5-6), 321-340, 2020.

Szántó, M., Oláh, A., Szöllősi, A., Tóth, K., Páyer, E., Czakó, N., Pór, Á., Kovács, I., Zouboulis, C., Kemény, L., Bíró, T., Tóth, I.: Activation of TRPV3 inhibits lipogenesis and stimulates production of inflammatory mediators in human sebocytes: a putative contributor to dry skin dermatoses.
J. Invest. Dermatol. 139 (1), 250-253, 2019.

Szántó, M., Dózsa, A., Antal, D., Szabó, K., Kemény, L., Bai, P.: Targeting the gut-skin axis - Probiotics as new tools for skin disorder management?.
Exp. Dermatol. 28 (11), 1210-1218, 2019.

Márton, J., Péter, M., Balogh, G., Bódi, B., Vida, A., Szántó, M., Bojcsuk, D., Jankó, L., Bhattoa, H., Gombos, I., Uray, K., Horváth, I., Török, Z., Bálint, B., Papp, Z., Vígh, L., Bai, P.: Poly(ADP-ribose) polymerase-2 is a lipid-modulated modulator of muscular lipid homeostasis.
Biochim. Biophys. Acta. Mol. Cell Biol. Lipids. 1863 (11), 1399-1412, 2018.

Oláh, A., Alam, M., Chéret, J., Kis, G., Hegyi, Z., Szántó, M., Bai, P., Lerchner, J., Bíró, T., Paus, R.: CB1 is a novel regulator of epidermal mitochondrial functions.
J. Invest. Dermatol. 137 (Suppl.), S210, 2017.

Abdul-Rahman, O., Kristóf, E., Doan-Xuan, Q., Vida, A., Nagy, L., Horváth, A., Simon, J., Maros, T., Szentkirályi, I., Palotás, L., Debreceni, T., Csizmadia, P., Szerafin, T., Fodor, T., Szántó, M., Tóth, A., Kiss, B., Bacsó, Z., Bai, P.: AMP-Activated Kinase (AMPK) Activation by AICAR in Human White Adipocytes Derived from Pericardial White Adipose Tissue Stem Cells Induces a Partial Beige-Like Phenotype.
PLoS One. 11 (6), e0157644, 2016.

Fodor, T., Szántó, M., Abdul-Rahman, O., Nagy, L., Dér, Á., Kiss, B., Bai, P.: Combined Treatment of MCF-7 Cells with AICAR and Methotrexate, Arrests Cell Cycle and Reverses Warburg Metabolism through AMP-Activated Protein Kinase (AMPK) and FOXO1.
PLoS One. 11 (2), 1-16, 2016.

Kiss, B., Szántó, M., Szklenár, M., Brunyánszki, A., Marosvölgyi, T., Sárosi, E., Remenyik, É., Gergely, P., Virág, L., Decsi, T., Rühl, R., Bai, P.: Poly(ADP) ribose polymerase-1 ablation alters eicosanoid and docosanoid signaling and metabolism in a murine model of contact hypersensitivity.
Mol. Med. Rep. 11 (4), 2861-2867, 2015.

Szántó, M., Bai, P.: Poly-(ADP-ribose) polymerases and mitochondria.
In: Mitochondrial Diseases and Therapy. Szerk.: V.Vijaya Padma, SM Group, Irving, 1-9, 2015.

Szántó, M., Brunyánszki, A., Márton, J., Vámosi, G., Nagy, L., Fodor, T., Kiss, B., Virág, L., Gergely, P., Bai, P.: Deletion of PARP-2 induces hepatic cholesterol accumulation and decrease in HDL levels.
Biochim. Biophys. Acta. Mol. Basis Dis. 1842 (4), 594-602, 2014.

Nagy, L., Docsa, T., Szántó, M., Brunyánszki, A., Hegedűs, C., Márton, J., Kónya, B., Virág, L., Somsák, L., Gergely, P., Bai, P.: Glycogen Phosphorylase Inhibitor N-(3,5-Dimethyl-Benzoyl)-N'-(β-D-Glucopyranosyl)Urea Improves Glucose Tolerance under Normoglycemic and Diabetic Conditions and Rearranges Hepatic Metabolism.
PLoS One. 8 (7), e69420-, 2013.

Szántó, M., Brunyánszki, A., Kiss, B., Nagy, L., Gergely, P., Virág, L., Bai, P.: Poly(ADP-ribose) polymerase-2: emerging transcriptional roles of a DNA-repair protein.
Cell. Mol. Life Sci. 69 (24), 4079-4092, 2012.

Bai, P., Cantó, C., Brunyánszki, A., Huber, A., Szántó, M., Cen, Y., Yamamoto, H., Houten, S., Kiss, B., Oudart, H., Gergely, P., Menissier-de, M., Schreiber, V., Sauve, A., Auwerx, J.: PARP-2 regulates SIRT1 expression and whole-body energy expenditure.
Cell Metab. 13 (4), 450-460, 2011.

Szántó, M., Rutkai, I., Hegedűs, C., Czikora, Á., Rózsahegyi, M., Kiss, B., Virág, L., Gergely, P., Tóth, A., Bai, P.: Poly(ADP-ribose) polymerase-2 depletion reduces doxorubicin-induced damage through SIRT1 induction.
Cardiovasc. Res. 92 (3), 430-438, 2011.

Brunyánszki, A., Hegedűs, C., Szántó, M., Erdélyi, K., Kovács, K., Schreiber, V., Gergely, S., Kiss, B., Szabó, É., Virág, L., Bai, P.: Genetic ablation of PARP-1 protects against oxazolone-induced contact hypersensitivity by modulating oxidative stress.
J. Invest. Dermatol. 130 (11), 2629-2637, 2010.